Workshop September 2nd and 3rd 2023 in Katowice, nr Krakow, Poland
Richard Holding DO has been in practice for over 50 years (qualified in 1969) and has specialised in the treatment of patients with chronic conditions that have resisted other approaches of treatment.
He has taught osteopathy in the cranial field internationally having studied and taught with Rolin Becker, Anne Wales, Harold Magoun, Robert Fulford, among others.
He introduced both Applied Kinesiology and Clinical Kinesiology as a Post Graduate subject at the then British School of Osteopathy in London UK in the 1980s.
He also taught Clinical Kinesiology internationally before developing a new approach using muscle testing in 1987.
- Introduction into the development of my approach to diagnosis and treatment.
- what are they?
- types of mitochondria
- normal physiological maintenance
- importance of conservation of elements of mitochondria
- factors affecting conservation
- what can go wrong?
- infection, malnutrition, trauma, genetic causes, lipid metabolism, glutamine damage to citric acid cycle
- endogenous retroviruses
- exogenous retroviruses
- Example one: practical demonstration with the diagnosis and treatment of a complex patient
- Viral damage and health
- Significant number of chronic health patterns are maintained by viruses hiding behind bacteria, fungi and parasites and these can be responsible for treatment failure in a lot of autoimmune disease patterns.
- In treating viral damage in patients, we can often overlook what is called the “cell danger response” where extracellular mitochondria are released into the body and they signal to other cells in the body that we are under attack. This initiates a shut down of energy production to initially “starve” the viruses of energy. It is common to find that often the patient has not switched this back on after the viral attack.
- Exogenous viruses, either from viral load directly or from vaccines indirectly, can activate the 5-8% of genetic DNA that is retroviral DNA human endogenous retroviruses or the 33% of our DNA that is retroviral-like genetic material.
- This can have very unfortunate unforeseen consequences in both the short and long term. We should all be inhibiting this process in all our patients.
- Persistent Spike Protein Syndrome
- Overview of the pathology
- Damage to cell membranes
- Damage to mitochondria
- Erythrocyte agglutination
- Spike protein receptors
- Glycosolation receptors
- ACE receptors
- Nicotine acetylcholine receptors in the brain
- Histamine receptors
- Oestrogen receptors
- Receptors affecting the conservation of NAD, a cofactor for over 500 enzymes in the body
- Treatment overview
- Reverse transcriptase inhibition
- Reduce vascular inflammation
- Dislodge spike proteins from their receptor sites
- Neutralise the toxicity of the spike proteins
- Dissolve the spike proteins
- Use of binding agents
- There will be a demonstration on how to diagnose and correct vascular inflammation from the spike protein in the COVID itself and its vaccines.
- Example Two: practical demonstration with the diagnosis and treatment of a complex patient
- Example Three: practical demonstration with the diagnosis and treatment of a complex patient
- Closing remarks
Price 500 euro
Venue: Pstragowa 11a, Katowice, Poland – Wysocki’s Clinic